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6

There's no way to be sure from such scant information, but it would quite possibly be this one: Autologous Tenocyte Implantation in Patients With Chronic Achilles Tendinopathy (ATI)


5

Source 3 is a book sponsered by Cyanotech Corporation claiming beneficial effects for the human health using Astaxanthin. The author is employee of Cyanotech Corporation. There is a small chapter regarding beneficial effects of Astaxanthin for the tennis elbow, but is is only referring to source 1. I did not find anymore studies referring to the effects of ...


4

I am sensing some mix up here. Your are asking about artificial tendons but your quote is stating about joint replacements. Basically your equating apples and oranges. Joint replacements or joint arthroplasties are used to treat severely destructed joints. In four cases of five the reason for joint destruction is osteoarthrosis in which the joint ...


4

Tendons don't usually get replaced because there is tendon repair surgery instead. Getting your tendon (or any body part for that matter) replaced by an artificial tendon usually takes longer to recover than a tendon repair surgery, and also leaves you with some lifetime limitations, such as not be able to lift weight and you will not be able to compete in ...


4

As you grow older, your tendons become more rigid and brittle. This is probably because of decrease in the water content of tendons, which is one of the reasons why tendons are flexible. This decrease in water content makes your tendons stiffer and weaker. They also become less tolerable of stress. This causes you to be more prone to tendon injuries, which ...


4

If the purpose is to prevent tendon injuries, then stretching is not really an effective solution. This article took a look at nearly 2000 articles in Embase and PubMed, and distilled down to 10 representative studies, none of which found that stretching was an effective method for preventing injuries. (On a side note, I have done previous searches on ...


3

That tendon is attached to palmaris longus muscles, a muscle that some people lack. There are absolutely no functional differences between people that have it and those who don't. Check the wikipedia article for more information: https://en.wikipedia.org/wiki/Palmaris_longus_muscle


3

Graham Chiu put it very well: "Where would you get a ligament from that isn't going to make you worse off?" We rely on our ligaments for the structural stability of our skeleton. This supersedes the integrity of our muscular attachments, although there are a few exceptions. Since they're attached to muscles, tendons are going to be more superficial (easier ...


3

Here's my (non professional take), from reading and listening to Jill Cook and Steven Low: TL;DR: Tendonitis/tendinosis are obsolete terms. If someone uses them, they are probably 10 or more years behind the times. The current one is tendinopathy. Tendinopathies provoke little or no inflammation, and we still have a long way to understand them. In ...


2

I think it is mainly due to the evidence indicating poor accuracy (sensitivity and specificity) of conventional MRI. It means that false-positive or false-negative findings may be detected in MRI of elbow (1): MR arthrography is more accurate than conventional MRI of the elbow at 3 T. [In 54 out of 79 patients, the diagnoses made on MRI and MR ...


2

Inflammatory chemicals released into an inflamed joint stimulate pain receptors even at rest: Proinflammatory cytokines such as tumor necrosis factor-alpha, interleukin-1beta, interleukin-6 and interleukin-17 are also mediators of pain by directly acting on the nociceptive system. Proportions of nociceptive sensory neurons express receptors for these ...


2

Sorry for the wait! To orient ourselves first trace the Ulnar Nerve distally to the hand from the Brachial Plexus (BP) and then review how Cubital Tunnel Syndrome ties in (it's neurological relation). Let me know if you have a more specific question (this is most commonly what is seen -- can go into other areas. Sorry for so many abbreviations - these ...


2

There is no data to suggest that is the case. The placebo controlled safety study https://www.nature.com/articles/s41514-017-0016-9 found no differences in musculoskeletal adverse side effects between active and control groups.


2

I found a 2017 article pointing specifically to a case of AAS usage associated with a Bilateral Patellar Tendon Rupture, which mentions: we suppose that combined AAS and exercise can increase tendon stiffness, predisposing to the rupture Additionally, one of this paper's sources references a 2016 study in which they point out that: Bilateral ...


2

Tendinopathy is a type of tendon disorder: Traditionally, tendons have been considered to be a mechanism by which muscles connect to bone as well as muscles itself, functioning to transmit forces. This connection allows tendons to passively modulate forces during locomotion, providing additional stability with no active work. The difference between non-...


1

Moshiri et al (2017) talks of Cissus quadrangularis with osteoporosis and mentions Cissus quadrangularis in relation to Osteobolin-C. Osteobolin-C is claimed to aid in the repair of joint, muscle, bone and connective tissue. The compounds found in osteobolin-C allow for the rapid mobilization of fibroblasts, chondroblasts and osteoblasts to the injured area,...


1

I performed a search of PubMed and found no studies for "cissus quadrangularis" and "tendinopathy". Looks like at this point there is no scientific evidence one way or another.


1

What you’ve heard is rooted in fact. CBD has well-known and well-characterized anti-inflammatory effects. [1,2] It also has demonstrable chondrogenic effects: This review discusses the role of the cannabinoid system in cartilage tissue and endeavors to establish if targeting the cannabinoid system has potential in mesenchymal stem cell based tissue-...


1

Your question focuses on the location of the receptors (the part of the nociceptors that is reacting to mechanical, thermal, or chemical stimuli). An equally important part of a nociceptor is the axon, or nerve fibre, that conducts the action potential from the muscle or tendon (to go with your example) all the way to the spinal cord from where it makes ...


1

Of course. Icing helps by numbing the pain sensation, thereby providing relief. If iced continuously it would lead to tissue damage with exact same effect of exposing your skin to below freezing temps. Frost bite but from within. Tried to ELI5 as much as possible. Hope it helps.


1

Depending on your age, clinical treatment with rest, physiotherapy, NSAID, epicondylitis, both lateral and medial, may take one to several months to resolve. Surgery is a last resort and should be reserved to those cases where there is complete rupture of a tendon.


1

The cite relates to joint replacement, a procedure of removing the painful joint surfaces and replacing them with an artificial joint (usually made of a soft synthetic material) which are placed in the ends of the bone – re-creating the joint that has been removed. The artificial surfaces of the joint replacement are shaped in such a way as to allow joint ...


1

You can use an ultrasound probe with power doppler to look at blood flow to see if there is increased blood flow present that would be compatible with an inflammatory process. And, neovascularization demonstrated by power Doppler may be present in more painful lesions http://pubs.rsna.org/doi/full/10.1148/radiol.2272012069 but currently it's not possible ...


1

The penetration is limited, therefore it's less risky than a steroid injection which carries a higher risk of tendon rupture(1). Study conclusion from 1999 (PDF) by SMJ was: Iontophoresis using sterile water or corticosteroid resulted in minimal or no biochemical and histologic changes in the tendon compared with injection of either substance. The method ...


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